Hypertension Goals

Hypertension, or high blood pressure, a major risk factor for cardiovascular diseases, significantly intersects with the United Nations' Sustainable Development Goals (SDGs). It is most directly linked with SDG 3 (Good Health and Well-being), underscoring the need for accessible and quality healthcare services for the detection, treatment, and control of hypertension. It also connects with SDG 2 (Zero Hunger), as balanced nutrition is vital in preventing and managing hypertension. Furthermore, the condition has indirect implications for SDG 1 (No Poverty) and SDG 10 (Reduced Inequalities), as the disease burden of hypertension often disproportionately affects low- and middle-income populations, reflecting and exacerbating existing socioeconomic inequalities. The awareness and prevention strategies necessary to combat hypertension further tie to SDG 4 (Quality Education), highlighting the importance of health literacy.

This Article supports SDG 3 by assessing ethnic differences in hypertension management. The findings suggest that people of African American Caribbean ethnicity had poorer blood pressure control due to stopping regular treatment.

Endocrine Hypertension
From Basic Science to Clinical Practice
2023, Pages 113-125

This content links with Goal 3: Good health and well-being and Goal 10: Reduced Inequalities by bringing recognition to Congenital adrenal hyperplasia (CAH), a group of monogenic, autosomal recessive disorders.
Relative number of participants with a diagnosis of hypertensive disorders of pregnancy (HDP) in studies that assessed its association with cardiovascular morbidity and mortality.
In studies of hypertensive disorders of pregnancy, white women are overrepresented. There is limited and heterogeneous reporting of race and ethnicity information across studies and few include race and/or ethnicity variables in statistical analysis.

Clinics in Chest Medicine, Volume 42, March 2021

Pulmonary arterial hypertension (PAH) occurs in women more than men whereas survival in men is worse than in women. In recent years, much research has been carried out to understand these sex differences in PAH. This article discusses clinical and preclinical studies that have investigated the influences of sex, serotonin, obesity, estrogen, estrogen synthesis, and estrogen metabolism on bone morphogenetic protein receptor type II signaling, the pulmonary circulation and right ventricle in both heritable and idiopathic pulmonary hypertension.

Illustration of process for haplotype-specific reporter construct derivation.
This Article supports SDG 3 by analysing data from four international cohorts of patients with pulmonary arterial hypertension, a disease caused by rare genetic variants.